Profile overview. Ryan D. Beveridge shows h-index 14 with 1,148 total citations across 26 indexed papers, averaging ~44 citations per paper β respectable for translational molecular biology, where citation density is lower than in genomics or AI. Duplicate entries in the record (e.g., the CRISPR-Cas9 oxidative phosphorylation paper, the NP105-113 COVID paper, and MRNIP/C5orf45 appear twice) suggest the true unique paper count is closer to ~22, meaning metrics may be slightly inflated by indexing duplication.
Research trajectory and strengths. His early-career work is anchored in DNA damage response and genome stability β centriolar satellite proteins (Cep131, CCDC13), the MRN complex interactor MRNIP/C5orf45, UCHL3/TDP1 proteostasis, and Fanconi anaemia pathway involvement in glioma. This body overlaps with large collaborative resources (genome-wide knockout mouse phenotyping). He then pivoted to AML/ALL leukemia genetics β MLL-AF4/KMT2A-AFF1 regulatory networks and a notable neomorphic progenitor paper on C/EBPΞ±/GATA-2 bilineage erythroid leukemia β before the 2020-2023 COVID era, where he co-authored mechanistic T cell work on proteasomal antigen processing of SARS-CoV-2 variants and an immunodominant HLA-B*07:02-restricted NP response linked to milder disease. Recent work on lentiviral vector production and low-pH CRISPR screens indicates applied bioprocessing and tumor microenvironment expansion.
Limitations and blindspots. No affiliation metadata was retrievable (OpenAlex returned a rate-limit error), so institutional context, grant portfolio, and h-index inflation from review articles cannot be independently verified. Citation counts are dominated by co-authored work; first/senior authorship positions are not visible from the metadata, and per-paper citation distributions (rather than totals) are unknown. The record lacks any single landmark paper with thousands of citations, which constrains the score.
Know what changed, what holds up, and what remains uncertain. Every Friday. No ads.